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>   首页   >   产品   >   一抗   >   基因重组   >   Anti-GOLM1 Reference Antibody (Cureab patent anti-GP73)   

Anti-GOLM1 Reference Antibody (Cureab patent anti-GP73)

Recombinant Antibody

     
  • 1 - Anti-GOLM1 Reference Antibody (Cureab patent anti-GP73) APR10920
    Anti-GOLM1 Reference Antibody (Cureab patent anti-GP73) on SDS-PAGE under reducing (R) condition. The gel was stained with Coomassie Blue. The purity of the protein is greater than 90%
  • 1 - Anti-GOLM1 Reference Antibody (Cureab patent anti-GP73) APR10920
    The purity of Anti-GOLM1 Reference Antibody (Cureab patent anti-GP73)is more than 95% ,determined by SEC-HPLC.
  • 产品详情
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Product Information
Application
  • Applications Legend:
  • E=ELISA
  • WB=Western Blotting
  • IHC=Immunohistochemistry
  • IHC-P=Immunohistochemistry (Paraffin)
  • IP=Immunoprecipitation
  • IF=Immunofluorescence
  • IC=Immunochemistry
  • ICC=Immunocytochemistry
  • FC=Flow Cytometry
  • DB=Dot Blot
FC, Kinetics, Animal Model
Primary Accession Q8NBJ4
Reactivity Human, Mouse
Clonality Monoclonal
Isotype IgG1
Calculated MW 45333 Da
Additional Information
Target/Specificity GOLM1
Endotoxin < 0.001EU/ µg,determined by LAL method.
Conjugation Unconjugated
Expression system CHO Cell
Format Purified monoclonal antibody supplied in PBS, pH6.0, without preservative.This antibody is purified through a protein A column.
Protein Information
Name GOLM1
Synonyms C9orf155, GOLPH2
Function Unknown. Cellular response protein to viral infection.
Cellular Location Golgi apparatus, cis-Golgi network membrane; Single-pass type II membrane protein. Note=Early Golgi. Cycles via the cell surface and endosomes upon lumenal pH disruption
Tissue Location Widely expressed. Highly expressed in colon, prostate, trachea and stomach. Expressed at lower level in testis, muscle, lymphoid tissues, white blood cells and spleen. Predominantly expressed by cells of the epithelial lineage. Expressed at low level in normal liver. Expression significantly increases in virus (HBV, HCV) infected liver. Expression does not increase in liver disease due to non-viral causes (alcohol-induced liver disease, autoimmune hepatitis) Increased expression in hepatocytes appears to be a general feature of advanced liver disease. In liver tissue from patients with adult giant- cell hepatitis (GCH), it is strongly expressed in hepatocytes-derived syncytial giant cells. Constitutively expressed by biliary epithelial cells but not by hepatocytes.
Research Areas

For Research Use Only. Not For Use In Diagnostic Procedures.

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